Download PDF by Deborah Finkel, Chandra A. Reynolds: Behavior Genetics of Cognition Across the Lifespan (Advances

By Deborah Finkel, Chandra A. Reynolds

Besides psychopathology, cognition has been one of many basic phenotypic focal issues of the sphere of habit genetics due to the fact that its inception. Francis Galton’s 1874 exam of eminent households in Britain was once one of the earliest makes an attempt to enquire no matter if cognitive achievements run in households. This quantity provides present methodologies for realizing cognitive skills that stream past the superseded nature vs. nurture paradigm. fresh advances in either assortment and statistical modeling of dual facts, really longitudinal dual information, make this a particularly valuable second to provide a piece that provides a set of the groundbreaking learn on cognitive talents around the lifespan. This quantity offers an outline of the present country of quantitative and molecular genetic investigations into the various elements of cognitive functionality and functioning around the lifespan.​

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Wadsworth, S. , DeFries, J. , & Hewitt, J. K. (2009). The developmental etiology of high IQ. Behavior Genetics, 39(4), 393–405. 1007/s10519-009-9268-x Butcher, L. , Davis, O. , Craig, I. , & Plomin, R. (2008). Genome-wide quantitative trait locus association scan of general cognitive ability using pooled DNA and 500K single nucleotide polymorphism microarrays. Genes Brain Behav, 7(4), 435–446. x Cardon, L. R. (1994). Specific cognitive abilities. R. P. J. C. DeFries & D. W.  57–76). Oxford: Blackwell.

20582 Light, J. , & DeFries, J. C. (1995). Comorbidity of reading and mathematics disabilities: genetic and environmental etiologies. Journal of Learning Disabilities, 28(2), 96–106. Loehlin, J. , & Nichols, R. C. (1976). Heredity, environment, and personality: A study of 850 sets of twins. Austin: University of Texas Press. Loo, S. , Doyle, A. , McGough, J. , et al. (2012). Genomewide association study of intelligence: Additive effects of novel brain expressed genes. Journal of the American Academy of Child and Adolescent Psychiatry, 51(4), 432–440.

1998; Petrill et al. 1997), results of this study indicated that during the transition to adolescence (after age 9), genetic factors contributed only to continuity (Fig. 5). Subsequent analyses extending to age 16 with data from only sibling pairs (Petrill et al. 2004) confirmed moderate-to-strong genetic and nonshared environmental influences at each age, and no evidence of new genetic influence at age 16. Similar to the findings of Bishop et al. (2003), genetic influences were found to be responsible for continuity and nonshared environmental influences were responsible for change.

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